By Andreas Moritz 

What actually happens when you eat meat?

The most blood-thickening agent is food protein, particularly if it is derived from an animal source. Let us assume you eat a medium-sized piece of steak, chicken or fish (cadaver protein). When compared to a carnivorous animal like a lion or a wolf, your stomach can produce only the relative amount of 1/20 of the hydrochloric acid needed to digest such a concentrated protein meal. In addition, the relative concentration of the hydrochloric acid in cats or wolves is at least five times higher than in humans. A cat or wolf can easily eat and digest the bones of a chicken, whereas humans cannot. Most of the cadaver’s protein, therefore, will pass undigested into the small intestine where it will either putrefy (80%) or enter the bloodstream (20%).

The liver is able to break down some of the absorbed protein, which forms the waste products urea and uric acid. This waste matter is passed on to the kidneys for excretion with the urine. However, with regular consumption of animal proteins, including meat, poultry, fish, eggs, cheese, and milk, more and more intrahepatic stones are formed in the bile ducts of the liver. This greatly reduces the liver’s ability to break down these proteins. (Stones in the bile ducts and gallbladder can be removed safely and painlessly through a series of liver flushes)

Protein foods are among the most acid-forming and blood-thickening foods of all. Therefore, when a major portion of the protein ends up circulating in the blood, it will, of course, thicken the blood. To avoid the danger of a heart attack or stroke, the body will attempt to dump the protein into the fluid surrounding the cells (tissue fluid or connective tissue). This thins the blood and averts the imminent danger of serious cardiovascular complications, at least for the time being. However, the dumped protein begins to turn the intercellular fluid into a gel-like substance. In this condition, nutrients that are trying to make their way to the cells may be caught in the thick soup, which increases the risk of cell death due to starvation.

How the body copes

The body tries to avoid cell death by initiating another, even more sophisticated, survival response, which is next to ingenious. To remove the proteins from the intercellular fluid, the body rebuilds the protein and converts it into collagen fiber, which is 100% protein. In this form, the body is able to build the protein into the basal membrane of the blood vessel walls. While accommodating the excessive protein, the basal membrane may become up to eight times as thick as is normal. Once, the capillary walls are saturated with the protein or collagen fiber, the basal membranes of the arteries start doing the same. This eventually, leads to a hardening of the arteries, which is the cause of heart disease, strokes and high cholesterol.

Now the body must face an even greater challenge. The thick walls of the capillaries (and possibly, the arteries) have become an obstruction, blocking the nutrient supply to the cells. The blood vessel walls increasingly prevent oxygen, glucose, and even water from penetrating the protein barricades, thus depriving cells of their bare nutrient essentials. Less glucose makes its way to the cells. As a result, cell metabolism drops to a lower level of efficiency, and waste production increases, similar to a car engine that has not been tuned properly or given quality gas or oil.

In addition to congesting the blood vessel walls, another complicating factor comes into play. Part of the excessive protein is absorbed by the lymphatic ducts that accompany every blood capillary. These lymph ducts and their attached lymph nodes are designed to remove and detoxify the normal amounts of cell-generated metabolic waste products. They also take away the cellular debris resulting from the daily destruction of over 30 billion worn-out cells. Since cells are composed of proteins, much of the collected waste is already filled with old cell protein. Being forced to take up extra protein from ingested foods like meat, fish, or milk simply overtaxes the entire lymphatic system and leads to the stagnation of lymph flow and fluid retention. Consequently, the congested lymph ducts are increasingly disabled as they attempt to take up the cells’ metabolic waste products. This, in turn, leads to a higher concentration of metabolic waste material in the fluid surrounding the cells.

Suffocation in Progress

The consequence of waste buildup in the cell environment is that cells not only become deprived of oxygen and other vital nutrients, but they also begin to suffocate in their own waste. The dramatic change of the cell environment leaves them with no other choice but to mutate into ‘abnormal’ cells,given the circumstances.

Cell mutation does not occur because the genes of the cell had a bad day and decided to play malignant. Genes do not switch themselves on and off without a reason. Genetic blueprints have no control or power to do anything. They are merely there to help the cell reproduce itself. However, the genetic blueprint becomes naturally altered when the environment of the cell undergoes major changes. By drastically reducing the concentration of oxygen in the cell environment, out of necessity the genes generate a new blueprint that enables them to survive without oxygen and instead use some of the metabolic waste products for energy. Mutated cells can take up, for example, lactic acid, and by metabolizing it, they are able to cover some of their energy needs. Although this abnormal type of cell metabolism has harmful side effects, by doing this, the body can avert, at least for a little while, the fatal poisoning of the affected organ or the blood. By keeping at least some of the oxygen-deprived cells alive through cell mutation, the organ is safeguarded against irreversible and sudden collapse and failure. All of these adaptations make cancer a survival mechanism to keep the person alive for as long as circumstances permit.

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This is an excerpt from my book CANCER IS NOT A DISEASE! – IT’S A SURVIVAL MECHANISM

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