By Andreas Moritz

When sugar becomes trapped and begins to increase in the blood stream, eating sugar at this point can be life-threatening. Not having enough glucose reaching the cells and organs of the body can also be fatal. If the heart cells run out of glucose, heart failure occurs. If the kidney cells run out of glucose, kidney failure occurs. If the eyes don’t get their glucose, eyesight will fail. The same applies to a sugar-starved liver, pancreas, stomach, brain, muscle, and even bone cells. By not receiving enough glucose, the body begins craving food, especially sugars, sweets, starchy foods, sweet beverages, etc., which leads to overeating and further congestion, and possibly heart congestion or cancer.

Because type II diabetes affects the health of every single one of the 60 trillion cells in the body, diabetics are predisposed to developing virtually every type of disorder there is. This has been denied by medical science for many years, but has recently been verified through major medical research. The majority of the chronic disorders plaguing our modern world today, including heart disease, cancer, arthritis, MS, Alzheimer’s, Parkinson’s, etc., may in actual fact not be separate diseases at all. While sharing the same cause or causes, they manifest themselves in different parts of the body as unique symptoms of disease. There will come a time when the practicing physician will recognize that diabetes, cancer, heart disease, and dementia, for example, share the same underlying causes, and therefore require the same treatment.

At the beginning stages of type II diabetes, the pancreas tries to respond to the increasing congestion of the blood vessel walls (with excessive proteins) and, possibly, to an excessive sugar or starch consumption, by secreting extra large amounts of insulin. By constantly producing disproportionate amounts of insulin, the cells become even further resistant to insulin. By blocking out insulin (along with vital nutrients) the cells try protecting themselves against the cell-damaging effects of too much insulin, or otherwise they would have to face cell mutation. Eventually, though, through intricate hormonal feedback mechanisms and enzyme signals, the pancreas recognizes both the increase in blood sugar levels and the shortage of cellular sugar, proteins and fatty acids. So the pancreas begins to deactivate, destroy or ‘put to sleep’ a large number of its insulin-producing cells. This practically sets the stage for non-insulin dependent diabetes to become insulin-dependent diabetes.

There are a number of other reasons that may lead to reduced insulin output by the pancreas. When the basal membranes of blood capillaries supplying the pancreas with nutrients become congested with protein fiber, insulin production and other important functions, such as production of digestive enzymes, become suppressed. The same occurs when stones in the bile ducts of the liver and gallbladder drastically reduce bile secretion. In an increasing number of individuals, bile sludge consisting of small cholesterol stones enters the common bile duct and gets caught up in the Ampulla of Vater (where the common bile duct and pancreatic duct meet). Bile activates pancreatic enzymes before they enter the small intestine to aid in the digestion of foods. If bile flow is restricted, not all of the enzymes dispatched by the pancreas are activated. Any of these unused enzymes remaining in the pancreas can damage or destroy pancreatic cells, which leads to pancreatitis – a common cause of diabetes and pancreatic cancer. In any case, the inability of the pancreas to produce enough insulin can be a lifesaver, at least temporarily.

It is obvious, though, that this act of cancer-preventive self-preservation also means that there is not enough insulin around to transport the sugar out of the blood stream. If type II diabetics become insulin-deficient, doctors often prescribe insulin in addition to blood sugar medication, while letting them continue eating protein foods. Thus, a previously non-insulin-dependent diabetic now needs insulin shots, which greatly increases his health risks. This is completely unnecessary. I have witnessed such insulin-dependent patients turn vegan, and within just six weeks become free of the main signs and symptoms of diabetes, for the first time in 20-30 years.

Chronic disease is only chronic for as long as its causes are still intact. Insulin injection is the very thing that keeps the patient from recovering. It continues to increase the cells’ resistance to insulin, and forces the pancreas to destroy an ever-increasing number of insulin-producing cells. There are plenty of natural things that can replace injection with insulin. Just one teaspoon of ground cinnamon per day can balance blood sugar. Turmeric is an amazing herb/spice with a similar effect. Broccoli and other vegetables, as well as regular full body exposure to sunlight (vitamin D-generating), have superior blood sugar-regulating effects than potentially dangerous insulin injections.

Researchers at the University of California-Los Angeles School of Medicine (UCLA) found that compared to subjects with the highest vitamin D levels, those with the lowest levels had symptoms of type II diabetes, including weaker pancreatic function and greater insulin resistance. When the skin is exposed to ultraviolet light, the body responds by manufacturing vitamin D.

Abstaining from proteins foods, cleansing the liver of stones (gallstones are a leading cause of diabetes), eating a balanced diet and living a balanced lifestyle are much more effective means of restoring normal body functions than just trying to fix one symptom of disease. By taking responsibility for their own health, and therefore their life, the diabetic has the opportunity to put the sweetness back into his cells and, thus, into their life.

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This is an excerpt from my book DIABETES – NO MORE!

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